No matter if you live in an apartment or a house or simply need to ensure that your house is free of electromagnetic fields, there are a number of ways you can reduce exposure. One of the most effective is to reduce the usage of your electronic devices. You could also consider EMF block paint to stop EMF radiation from entering your home. Another easy way to protect your house from EMF radiation would be to install an RF shielding cover. This is a cloth of net that contains EMF shielding. It's used to block EMFs from entering a room. Another option is to have your home fitted with an enclosure that is conductive. These devices are called Faraday cages.
Several studies have shown studies have shown that the nonionizing EMF produces anti-proliferative effects on HCC cells. The original source behind AM RF EMF's anticancer activity in vitro is believed be based on the downregulation of cancer stem cells. This may account for the long-term effects observed in patients with advanced HCC. However, Website link of AM EMF's impact on cancer patients is not evident.
Aspects from AM RF EMFs on HCC tumour growth in vivo were studied in mice. The tumours were separated in three different groups. First, the group that was unaffected RF EMF. Second group members were exposed RF EMF at the same frequency to that of humans. The third group was exposed the RF EMF in HCC-specific frequencies. The impact of HCCMF on tumours was evaluated against that of RCF. The results showed that the tumors treated by HCCMF had significant shrinkage. However, the tumours treated with RCF showed no evidence of shrinkage in the tumour.
The reason for tumour-specific AM RF EMF might be driven by the fact that tumor cells require Cav3*2 type voltage calcium channels to promote proliferation and down-regulation. AM RF EMF's ability to inhibit proliferation on HCC cells is caused through CACNA1H, a protein that regulates the Ca2+ influx specific to tumors. The results indicate that CACNA1H could have wider implications for diagnosis and treatment of various cancers.
The tumors in the control group were not exposed EMF from radiofrequency, and fed a normal mouse diet. emf blocking of the HCCMF group were injected with Huh7 cells when they were between five and seven weeks old. The tumors were removed when they showed excessive burden.
The tumors of the three groups also showed different growth curves. The tumours treated with HCCMF had a significant reduction in size of the tumor after eight weeks. However, tumors treated with RCF didn't show reduction in size. The difference was significant. The tumors treated with RCF showed necrosis, which is typical in tumors that have been that are exposed to RCF. It is possible that the necrosis was caused by an absence of oxygen in the more invasive cancers.
In sum, the results indicate that AM RF EMF has anticancer activity in vitro and in the vivo. Numerous studies have demonstrated that AM RF EMF produces measurable tumour shrinkage in HCC patients. The possibility is that the AM EMF causes these effects through CACNA1H which is a protein involved in the tissue-specific Ca2+ influx. Additionally, AM RF EMF may have a long-lasting impact on the growth of HCC tumours in vivo.